1. Indications for treatment: If the wound heals within 10 to 14 days, a prophylactic elastic sleeve can be applied to patients with black scars. If repair takes 14 to 21 days, all patients should undergo prophylactic compression therapy. If the redness subsides within 2 to 3 months, it can be concluded that the scar will gradually improve and no compression therapy is required. If it is still highly vascularized, bright red in color, and progressively hardened after 2 months, it means that scarring will form, and compression therapy must be continued. If the repair is more than 21 days, pressure therapy must be applied long-term.

2. The key to the success of compression therapy lies in "early", "tight" and "long". After the wound has healed, pressure should be started as soon as possible. After the scar is obvious, the pressure effect is not good. The pressure is 2.13kPa -2. 39kPa is appropriate. If the elastic sleeve becomes loose, it will lose its pressure function and must be replaced in time. If the patient feels too tight and cannot tolerate it, he can relax a little to reduce the pressure, but the elastic sleeve cannot be released. The elastic sleeve must be worn 18 hours to 24 hours a day for at least 4 months to 6 months, and even up to 2 years. Relaxing prematurely may cause the scar to spring back. Generally, after 4 months to 6 months of continuous pressure, the scar congestion disappears and the swelling becomes flat. After 8 months to 10 months, the pressure can be terminated in most cases. whitened.

3. The mechanism of the histopathological changes of compression therapy is not clear, it may be that pressure causes ischemia, activates tissue metabolism, and increases collagenase activity. Due to hypoxia-ischemia, the number of tubes is reduced, the blood supply to scar tissue is reduced, and the partial pressure of oxygen in cells is reduced under hypoxia. Mitochondrial function declines or even stops. Morphological changes occurred at the same time. Such as mitochondrial swelling, vacuolar degeneration and so on. In this way, mitochondria, which are mainly used in cell biological oxidation, cannot release energy well in a series of oxidative phosphorylation processes, resulting in inhibited proliferation of fibroblasts, and finally degeneration and necrosis. The function of generating collagen fibers and matrix is greatly reduced. This results in a thinning and softening of the scar.